RASSF10 is frequently epigenetically inactivated in kidney cancer and its knockout promotes neoplasia in cancer prone mice

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dc.contributor.authorRichter, Antje M.
dc.contributor.authorWoods, Michelle L.
dc.contributor.authorKüster, Miriam M.
dc.contributor.authorWalesch, Sara K.
dc.contributor.authorBraun, Thomas
dc.contributor.authorBoettger, Thomas
dc.contributor.authorDammann, Reinhard H.
dc.date.accessioned2022-08-22T13:57:17Z
dc.date.available2022-08-22T13:57:17Z
dc.date.issued2020
dc.identifier.urihttps://jlupub.ub.uni-giessen.de//handle/jlupub/6665
dc.identifier.urihttp://dx.doi.org/10.22029/jlupub-6116
dc.language.isoen
dc.rightsNamensnennung 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.subject.ddcddc:570
dc.titleRASSF10 is frequently epigenetically inactivated in kidney cancer and its knockout promotes neoplasia in cancer prone mice
dc.typearticle
local.affiliationFB 08 - Biologie und Chemie
local.source.epage3127
local.source.journaltitleOncogene
local.source.spage3114
local.source.urihttps://doi.org/10.1038/s41388-020-1195-6
local.source.volume39

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