Epigenetically silenced apoptosis-associated tyrosine kinase (AATK) facilitates a decreased expression of Cyclin D1 and WEE1, phosphorylates TP53 and reduces cell proliferation in a kinase-dependent manner

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dc.contributor.authorWoods, Michelle L.
dc.contributor.authorWeiss, Astrid
dc.contributor.authorSokol, Anna M.
dc.contributor.authorGraumann, Johannes
dc.contributor.authorBoettger, Thomas
dc.contributor.authorRichter, Antje M.
dc.contributor.authorSchermuly, Ralph T.
dc.contributor.authorDammann, Reinhard H.
dc.date.accessioned2022-11-16T06:38:33Z
dc.date.available2022-11-16T06:38:33Z
dc.date.issued2022
dc.identifier.urihttps://jlupub.ub.uni-giessen.de//handle/jlupub/8484
dc.identifier.urihttp://dx.doi.org/10.22029/jlupub-7872
dc.language.isoen
dc.rightsNamensnennung 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.subject.ddcddc:610
dc.titleEpigenetically silenced apoptosis-associated tyrosine kinase (AATK) facilitates a decreased expression of Cyclin D1 and WEE1, phosphorylates TP53 and reduces cell proliferation in a kinase-dependent manner
dc.typearticle
local.affiliationFB 11 - Medizin
local.source.epage1987
local.source.journaltitleCancer gene therapy
local.source.spage1975
local.source.urihttps://doi.org/10.1038/s41417-022-00513-x
local.source.volume29

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